Why H1N1 Likely Will NEVER Be a True Pandemic Threat
To finish up, I’d like to quote Peter Palese, Professor and Chair, Microbiology and Professor of Medicine, Infectious Diseases, Mount Sinai School of Medicine – one of the panel members of the Council On Foreign Relations Symposium on Pandemic Influenza:
“ … So, clearly, when I say this 2009 virus is like another seasonal virus, I don't mean this is a harmless virus which we should ignore. No. The regular flu is bad enough and this 2009 virus is also bad enough. Having said that, it lacks certain signatures, certain molecular signs which are associated with the 1918 pandemic and then explained in 1957 and in 1968, and that is one of the genes.
… It has a technical name, PB1F2, and that is missing in the 2009 virus. And it could acquire this by exchanging that mini-chromosome and getting one which carries that PB1F2 or by point mutation, however, and that would make it more virulent.
However, it is sort of like if I put a more powerful engine into a VW, if I put a Lamborghini engine into a VW or a little Fiat or some car, it may not be better. It may not fit. It may not mesh and that, I think, may also be happening with the 2009.
By accumulating and getting mutations or getting this other gene, more virulent gene, it may not end up as something which is really a sports car which runs 200 miles an hour, so there is a lot which has to sort of fit and mesh in order to make a virus really a 1918 or 1957.”
Folks, from every which angle, the swine flu pandemic is NOT what it’s being made out to be. Perhaps it’s all about greed and POWER - the government and corporations controlling what gets into your body and what goes into their bank accounts; about making a few bucks in a suffering economy when people can’t afford their regular medications and sales are down.
Posted by: Dr. Mercola
November 17 2009 | 74,291 views
Tuesday, November 17, 2009
Friday, November 13, 2009
Satuday Breakfast at Harry and Sandie's
EXCITED
I just want to share how excited I am about all I'm learning from Cory about network marketing and blogging and the whole emarketing thing. He really is a great guy. He comes over at his own expense sometimes and puts out a great break fast every Saturday Morning 9am at our home 3046 Glenrosa( should be easy to find now I have house numbers). All the while we are able to become more informed by the doc talk CONFERENCE CALL about these wonderful Max products. Cory then set a side extra time to teach us to blog. By doing these blogs we have been able to capture extra income from google ad sense . I mean yesterday I couldn't beleive it I made .95 cents already never miNd all the leads we CAPTURE through the internet.
You know OPPORTUNITIES like this will only pass through lives maybe once or twice, so please take the time to come over enjoy a comfortable home atmosphere with myself HAPPY HARRY, SILENT SANDIE AND CALAMITY CORY.
I just want to share how excited I am about all I'm learning from Cory about network marketing and blogging and the whole emarketing thing. He really is a great guy. He comes over at his own expense sometimes and puts out a great break fast every Saturday Morning 9am at our home 3046 Glenrosa( should be easy to find now I have house numbers). All the while we are able to become more informed by the doc talk CONFERENCE CALL about these wonderful Max products. Cory then set a side extra time to teach us to blog. By doing these blogs we have been able to capture extra income from google ad sense . I mean yesterday I couldn't beleive it I made .95 cents already never miNd all the leads we CAPTURE through the internet.
You know OPPORTUNITIES like this will only pass through lives maybe once or twice, so please take the time to come over enjoy a comfortable home atmosphere with myself HAPPY HARRY, SILENT SANDIE AND CALAMITY CORY.
Saturday, October 31, 2009
MaxGxl Victoria
"Arthritis & Glutathione"
Arthritis is a group of conditions involving damage to the joints of the body. Arthritis is the leading cause of disability in people older than fifty-five years. There are different forms of arthritis; each has a different cause. The most common form of arthritis, osteoarthritis (degenerative joint disease) is a result of trauma to the joint, infection of the joint, or age. Emerging evidence suggests that abnormal anatomy might contribute to the early development of osteoarthritis.
Other arthritis forms are rheumatoid arthritis and psoriatic arthritis, autoimmune diseases in which the body attacks itself. Septic arthritis is caused by joint infection. Gouty arthritis is caused by deposition of uric acid crystals in the joint, causing inflammation. There is also an uncommon form of gout caused by the formation of rhomboid crystals of calcium pyrophosphate. This gout is known as pseudogout."
Arthritis is a group of conditions involving damage to the joints of the body. Arthritis is the leading cause of disability in people older than fifty-five years. There are different forms of arthritis; each has a different cause. The most common form of arthritis, osteoarthritis (degenerative joint disease) is a result of trauma to the joint, infection of the joint, or age. Emerging evidence suggests that abnormal anatomy might contribute to the early development of osteoarthritis.
Other arthritis forms are rheumatoid arthritis and psoriatic arthritis, autoimmune diseases in which the body attacks itself. Septic arthritis is caused by joint infection. Gouty arthritis is caused by deposition of uric acid crystals in the joint, causing inflammation. There is also an uncommon form of gout caused by the formation of rhomboid crystals of calcium pyrophosphate. This gout is known as pseudogout."
Wednesday, October 14, 2009
Energy Production
The Role of Glutathione in
Energy Production
You’ve been told that glutathione increases energy on a cellular level. How does it do this? Since glutathione is not the actual fuel for the “engines” or “energy factories” of our cells, what part does it play in the creation of cellular energy?
These are questions we would all like answers to, but not if they make us feel like we’re back in Chemistry 101! Hopefully, what you read below will provide a non- technical explanation for these questions.
The Tiger in Your Tank Is Called ATP
It’s obvious that all cells must produce energy to survive. We know that the“gasoline” or “electricity” that powers our cells is called ATP (adenosine triphosphate). If your car runs out of gas, it can sit for a while until you replenish the supply. Not so with our cellular fuel. Our cells simply cannot survive unless they have a continual, sufficient supply of “cellular gasoline” or ATP. So the process of creating fuel in each and every cell must go on constantly.
A single cell may have a couple of hundred energy factories or cellular power plants called mitochondria or it may contain thousands of them. Your hard-working heart cells have been estimated to have 5,000 mitochondria each1! The complex process of turning nutrients into ATP in the mitochondria depends on many oxidation and reduction reactions.
Redox is the shorthand term for the chemical reaction where atoms are changed by either losing elections (oxidation) or gaining electrons (reduction).
The Process
Understanding the exact process of ATP creation didn’t happen until 1937. Hans A. Krebs finally figured out a metabolic pathway in the cells that explained how energy is created. There were many scientists who played a key role in understanding how this pathway worked, but because Krebs had the insight that the pathway was a cycle, the process was named for him and is usually called the Krebs cycle.
You will also read of it as the “citric acid cycle” or the tricarboxylic acid cycle (TCA) or occasionally, the Szent-Györgyi-Krebs cycle (adding the name of the scientist who mapped out many of the molecules and the sequence of reactions of the Krebs cycle).
We are including a diagram of the eight major steps in the energy cycle. Since we are not doing Chemistry 101, describing this process could be as simple as saying it’s a metabolic dance of electrons being transported and transferred, through complex processes of reduction and oxidation. Remember that transferring (or grabbing!) electrons is also what starts the cycle of free radical damage.
The Role of Glutathione
Oxidation is like a flame, wonderful when harnessed, dangerous when out of control. When we are young, the oxidation necessary for energy production is highly efficient with only a small amount of oxygen not utilized. Even this minor oxygen loss (1-3%) unfortunately results in the production of free radicals. And this is where glutathione comes in to neutralize those free radicals.
A one-to-three percent oxygen loss causing free-radical production doesn’t sound too bad, does it? But say you are no longer 20 years of age and correspondingly, your cellular mitochondria are not as efficient as they once were. More and more free radicals are generated in the oxidative energy process and they are, in turn, damaging the mitochondria themselves.
The powerful antioxidant, glutathione, quenches a number of free radicals, including the highly dangerous hydroxyl radical by donating an electron. Glutathione itself does not become a free radical even though it has given up an electron.
No wonder it is called the Master Antioxidant!
Now that it is quenched, the extremely harmful hydroxyl radical is halted from its real dirty work: generating other free radicals like peroxynitrite and the radicals of oxidized fats (lipids).
Glutathione has just halted a literal cascade of free radicals by quenching the hydroxyl free radical. But you need to know just what a favor glutathione has done for you in preventing peroxynitrite production.
Peroxynitrite is truly pernicious!
Martin L. Pall, PhD, professor of Biochemistry and Basic Medical Science at Washington State University stated: “Peroxynitrite reacts with and inactivates several of the enzymes in mitochondria so that mitochondrial and energy metabolism dysfunction is one of the most important consequences of elevated
peroxynitrite.”2
Translation: When the mitochondria have suffered enough free radical damage as to become dysfunctional, energy production is impaired. Your cells do not
get the amount of energy they need to function properly. Your cells may
function at a level below the optimum although you probably won’t feel it.
There is yet one more free-radical mitochondrial consequence as a result of low glutathione levels that you “will” be aware of. Richard Van Konynenburg, PhD, wrote: “The resulting partial blockades in the Krebs cycles and the respiratory chains in the red, slow-twitch skeletal muscle cells decrease their rate of
production of ATP. Since ATP is what powers muscle contractions, the lack of it produces physical fatigue. It becomes chronic because GSH [glutathione] remains depleted.”3
You might think of free radicals as a band of marauders circling an oil tanker, preparing to hijack the precious commodity that fuels cars and trains and pretty much the entire Western world. Glutathione molecules could be considered a group of highly efficient bodyguards—handing out electrons and
turning vicious marauders into mild-mannered bystanders.
Glutathione Praise from Dr. Keller
As you understand this intricate energy creation process, perhaps you
will appreciate more a statement by the creator of MaxGXL, Dr. Robert H.
Keller. Dr. Keller said that glutathione is “critical to cell functions such as the
TCA (Kreb) cycle and then the respiratory chain that uses 4 molecules of ATP
to generate 32 molecules of ATP.”
Is it vital for you to have those glutathione bodyguards on energy duty? Van Konynenburg certainly thinks so as he wrote, “GSH [glutathione] plays very important roles in maintaining mitochondrial function and integrity.”4
Perhaps it’s enough to remember that glutathione is there to assure that the
creation of cellular energy is not hampered by free radical damage. While this is not by any means the only role glutathione plays in keeping your body running smoothly, it is a vitally important role. Now you have one more reason to appreciate Dr. Keller’s marvelous, patented product!
Notes
1. Stephen T. Sinatra, MD, FACC, “L-Carnitine and the Heart,” McGraw-Hill, 1999, p. 36
2. Pall M, “Elevated, sustained peroxynitrite levels as the cause of chronic fatigue syndrome,” Medical Hypotheses (2000) 54(1):115-125.
3. Glutathione White Paper, Richard Van Konynenburg, PhD, http://phoenix-cfs.org/ GluAACFS04.htm
4. Ibid.
Energy Production
You’ve been told that glutathione increases energy on a cellular level. How does it do this? Since glutathione is not the actual fuel for the “engines” or “energy factories” of our cells, what part does it play in the creation of cellular energy?
These are questions we would all like answers to, but not if they make us feel like we’re back in Chemistry 101! Hopefully, what you read below will provide a non- technical explanation for these questions.
The Tiger in Your Tank Is Called ATP
It’s obvious that all cells must produce energy to survive. We know that the“gasoline” or “electricity” that powers our cells is called ATP (adenosine triphosphate). If your car runs out of gas, it can sit for a while until you replenish the supply. Not so with our cellular fuel. Our cells simply cannot survive unless they have a continual, sufficient supply of “cellular gasoline” or ATP. So the process of creating fuel in each and every cell must go on constantly.
A single cell may have a couple of hundred energy factories or cellular power plants called mitochondria or it may contain thousands of them. Your hard-working heart cells have been estimated to have 5,000 mitochondria each1! The complex process of turning nutrients into ATP in the mitochondria depends on many oxidation and reduction reactions.
Redox is the shorthand term for the chemical reaction where atoms are changed by either losing elections (oxidation) or gaining electrons (reduction).
The Process
Understanding the exact process of ATP creation didn’t happen until 1937. Hans A. Krebs finally figured out a metabolic pathway in the cells that explained how energy is created. There were many scientists who played a key role in understanding how this pathway worked, but because Krebs had the insight that the pathway was a cycle, the process was named for him and is usually called the Krebs cycle.
You will also read of it as the “citric acid cycle” or the tricarboxylic acid cycle (TCA) or occasionally, the Szent-Györgyi-Krebs cycle (adding the name of the scientist who mapped out many of the molecules and the sequence of reactions of the Krebs cycle).
We are including a diagram of the eight major steps in the energy cycle. Since we are not doing Chemistry 101, describing this process could be as simple as saying it’s a metabolic dance of electrons being transported and transferred, through complex processes of reduction and oxidation. Remember that transferring (or grabbing!) electrons is also what starts the cycle of free radical damage.
The Role of Glutathione
Oxidation is like a flame, wonderful when harnessed, dangerous when out of control. When we are young, the oxidation necessary for energy production is highly efficient with only a small amount of oxygen not utilized. Even this minor oxygen loss (1-3%) unfortunately results in the production of free radicals. And this is where glutathione comes in to neutralize those free radicals.
A one-to-three percent oxygen loss causing free-radical production doesn’t sound too bad, does it? But say you are no longer 20 years of age and correspondingly, your cellular mitochondria are not as efficient as they once were. More and more free radicals are generated in the oxidative energy process and they are, in turn, damaging the mitochondria themselves.
The powerful antioxidant, glutathione, quenches a number of free radicals, including the highly dangerous hydroxyl radical by donating an electron. Glutathione itself does not become a free radical even though it has given up an electron.
No wonder it is called the Master Antioxidant!
Now that it is quenched, the extremely harmful hydroxyl radical is halted from its real dirty work: generating other free radicals like peroxynitrite and the radicals of oxidized fats (lipids).
Glutathione has just halted a literal cascade of free radicals by quenching the hydroxyl free radical. But you need to know just what a favor glutathione has done for you in preventing peroxynitrite production.
Peroxynitrite is truly pernicious!
Martin L. Pall, PhD, professor of Biochemistry and Basic Medical Science at Washington State University stated: “Peroxynitrite reacts with and inactivates several of the enzymes in mitochondria so that mitochondrial and energy metabolism dysfunction is one of the most important consequences of elevated
peroxynitrite.”2
Translation: When the mitochondria have suffered enough free radical damage as to become dysfunctional, energy production is impaired. Your cells do not
get the amount of energy they need to function properly. Your cells may
function at a level below the optimum although you probably won’t feel it.
There is yet one more free-radical mitochondrial consequence as a result of low glutathione levels that you “will” be aware of. Richard Van Konynenburg, PhD, wrote: “The resulting partial blockades in the Krebs cycles and the respiratory chains in the red, slow-twitch skeletal muscle cells decrease their rate of
production of ATP. Since ATP is what powers muscle contractions, the lack of it produces physical fatigue. It becomes chronic because GSH [glutathione] remains depleted.”3
You might think of free radicals as a band of marauders circling an oil tanker, preparing to hijack the precious commodity that fuels cars and trains and pretty much the entire Western world. Glutathione molecules could be considered a group of highly efficient bodyguards—handing out electrons and
turning vicious marauders into mild-mannered bystanders.
Glutathione Praise from Dr. Keller
As you understand this intricate energy creation process, perhaps you
will appreciate more a statement by the creator of MaxGXL, Dr. Robert H.
Keller. Dr. Keller said that glutathione is “critical to cell functions such as the
TCA (Kreb) cycle and then the respiratory chain that uses 4 molecules of ATP
to generate 32 molecules of ATP.”
Is it vital for you to have those glutathione bodyguards on energy duty? Van Konynenburg certainly thinks so as he wrote, “GSH [glutathione] plays very important roles in maintaining mitochondrial function and integrity.”4
Perhaps it’s enough to remember that glutathione is there to assure that the
creation of cellular energy is not hampered by free radical damage. While this is not by any means the only role glutathione plays in keeping your body running smoothly, it is a vitally important role. Now you have one more reason to appreciate Dr. Keller’s marvelous, patented product!
Notes
1. Stephen T. Sinatra, MD, FACC, “L-Carnitine and the Heart,” McGraw-Hill, 1999, p. 36
2. Pall M, “Elevated, sustained peroxynitrite levels as the cause of chronic fatigue syndrome,” Medical Hypotheses (2000) 54(1):115-125.
3. Glutathione White Paper, Richard Van Konynenburg, PhD, http://phoenix-cfs.org/ GluAACFS04.htm
4. Ibid.
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